Trans-splicing of a voltage-gated sodium channel is regulated by nerve growth factor

FEBS Lett. 1999 Feb 19;445(1):177-82. doi: 10.1016/s0014-5793(99)00126-x.

Abstract

Mammalian sensory neurons express a voltage-gated sodium channel named SNS. Here we report the identification of an SNS transcript (SNS-A) that contains an exact repeat of exons 12, 13 and 14 encoding a partial repeat of domain II. Because the exons 12-14 are present in single copies in genomic DNA, the SNS-A transcript must arise by trans-splicing. Nerve growth factor, which regulates pain thresholds, and the functional expression of voltage-gated sodium channels increases the levels of the SNS-A transcript several-fold both in vivo and in vitro as measured by RNase protection methods, as well as RT-PCR. These data demonstrate a novel regulatory role for the nerve growth factor and are the first example of trans-splicing in the vertebrate nervous system.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alternative Splicing
  • Animals
  • Cells, Cultured
  • DNA-Binding Proteins / genetics*
  • Gene Expression Regulation
  • Ion Channel Gating*
  • Nerve Growth Factors / metabolism*
  • Nerve Growth Factors / pharmacology
  • Neurons / cytology
  • Neurons / drug effects
  • Neurons / metabolism*
  • Rats
  • Rats, Sprague-Dawley
  • Snail Family Transcription Factors
  • Trans-Splicing*
  • Transcription Factors / genetics*

Substances

  • DNA-Binding Proteins
  • Nerve Growth Factors
  • Snai2 protein, rat
  • Snail Family Transcription Factors
  • Transcription Factors