Fas (APO-1/CD95) signaling pathway is intact in radioresistant human glioma cells

Cancer Res. 1999 Mar 15;59(6):1362-5.

Abstract

Radiation-induced apoptosis can be mediated through pathways initiated by either DNA damage or ceramide-induced Fas signaling. Glioblastoma multiforme is a primary brain tumor that is highly resistant to irradiation, and U-87 MG, SF126, and T98G are glioblastoma-derived cell lines that mimic this characteristic. We found that these radioresistant glioma cells are susceptible to Fas-mediated cell death induced by treatment with either anti-Fas antibody or exogenous ceramide. Fas-mediated cell death in these cell lines is p53-independent. These data demonstrate that apoptosis can be induced by ceramide and mediated through the Fas pathway in glioma cells, although high-dose ionizing radiation fails to trigger this pathway.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antibodies / pharmacology
  • Apoptosis*
  • Brain Neoplasms / metabolism*
  • Brain Neoplasms / pathology
  • Ceramides / pharmacology
  • Glioma / metabolism*
  • Glioma / pathology
  • Humans
  • Radiation Tolerance*
  • Signal Transduction* / drug effects
  • Signal Transduction* / radiation effects
  • Tumor Cells, Cultured
  • fas Receptor / immunology
  • fas Receptor / metabolism*

Substances

  • Antibodies
  • Ceramides
  • fas Receptor