Decreased sensory neuropeptide release from trachea of rats with streptozotocin-induced diabetes

Eur J Pharmacol. 1999 Mar 19;369(2):221-4. doi: 10.1016/s0014-2999(99)00089-8.

Abstract

We studied the release of somatostatin, calcitonin gene-related peptide (CGRP) and substance P in response to electrical field stimulation from isolated tracheas of rats following 4 weeks of streptozotocin (50 mg/kg i.v.)-induced diabetes. Field stimulation (40 V, 0.1 ms, 10 Hz for 120 s) increased the release of somatostatin, CGRP and substance P from the baseline 0.18+/-0.029, 0.17+/-0.027, and 1.77+/-0.086 to 0.51+/-0.022, 0.69+/-0.115, and 5.96+/-0.377 in control preparations and 0.31+/-0.081, 0.41+/-0.142, and 3.14+/-0.443 fmol/mg wet tissue weight in preparations from diabetic rats as measured by radioimmunoassay (control vs. diabetic P<0.01 for each). The results show a simultaneous decrease in release of the three sensory neuropeptides and an enhanced plasma somatostatin level in rats with streptozotocin-induced diabetes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blood Glucose / drug effects
  • Body Weight / drug effects
  • Calcitonin Gene-Related Peptide / blood
  • Calcitonin Gene-Related Peptide / metabolism
  • Diabetes Mellitus, Experimental / metabolism*
  • Electric Stimulation
  • In Vitro Techniques
  • Male
  • Neuropeptides / metabolism*
  • Radioimmunoassay
  • Rats
  • Rats, Wistar
  • Somatostatin / blood
  • Somatostatin / metabolism*
  • Substance P / blood
  • Substance P / metabolism
  • Trachea / metabolism*

Substances

  • Blood Glucose
  • Neuropeptides
  • Substance P
  • Somatostatin
  • Calcitonin Gene-Related Peptide