Aging-associated down-regulation of ClC-1 expression in skeletal muscle: phenotypic-independent relation to the decrease of chloride conductance

FEBS Lett. 1999 Apr 16;449(1):12-6. doi: 10.1016/s0014-5793(99)00202-1.

Abstract

In order to clarify the mechanism underlying the reduction of resting membrane chloride conductance (gCl) during aging, the levels of mRNA encoding the principal skeletal muscle chloride channel, ClC-1, were measured. Total RNA samples isolated from tibialis anterior muscles of aged (24-29 months old) and adult (3-4 months old) rats were examined for ClC-1 expression using Northern blot analysis, and macroscopic gCl was recorded from extensor digitorum longus muscle fibers from each adult and aged rat in vitro using a two intracellular microelectrode technique. Although interindividual variability was observed, aged rats exhibited a parallel reduction of both gCl and ClC-1 mRNA expression as compared to adult rats. A linear correlation exists between individual values of ClC-1 mRNA and gCl. These results provide evidence that ClC-1 is the main determinant of sarcolemmal gCl and demonstrate that the decrease of gCl observed during aging is associated with a down-regulation of ClC-1 expression in muscle.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging / physiology*
  • Animals
  • Chloride Channels / genetics*
  • Chlorides / metabolism
  • Chlorides / physiology
  • Down-Regulation*
  • Humans
  • Muscle Fibers, Skeletal / metabolism
  • Muscle Proteins / genetics*
  • Muscle, Skeletal / metabolism*
  • Muscle, Skeletal / physiology
  • Phenotype
  • Proteins / metabolism
  • RNA / metabolism
  • Rats
  • Rats, Wistar

Substances

  • CLC-1 channel
  • Chloride Channels
  • Chlorides
  • Muscle Proteins
  • Proteins
  • RNA