Killing of T lymphocytes by synthetic ceramide is by a nonapoptotic mechanism and is abrogated following mitogenic activation

Exp Cell Res. 1999 May 25;249(1):116-22. doi: 10.1006/excr.1999.4474.

Abstract

Ceramide induces apoptosis in leukemia cell lines and has been proposed as a potential therapeutic agent in malignancies refractory to conventional treatment. Here we show that synthetic N-acetyl-d-erythro-sphingosine (C2 ceramide) kills normal human T lymphocytes by a caspase-independent nonapoptotic mechanism. By contrast, T cells were induced to caspase-dependent apoptosis by okadaic acid. Furthermore, C2 ceramide treatment of the Jurkat leukemia cell line induced killing by apoptosis. Activation of T lymphocytes by phytohemagglutinin abrogated killing by C2 ceramide. The data here suggest that ceramide triggers caspase-dependent apoptosis in leukemia cells lines, but activates caspase-independent nonapoptotic killing of resting T lymphocytes which is abrogated following mitogenic activation.

Publication types

  • Comparative Study

MeSH terms

  • Amino Acid Chloromethyl Ketones / pharmacology
  • Apoptosis / drug effects
  • Caspases / metabolism
  • Cell Death
  • Cells, Cultured
  • Cysteine Proteinase Inhibitors / pharmacology
  • Enzyme Activation / drug effects
  • Humans
  • Jurkat Cells / drug effects
  • Lymphocyte Activation / drug effects
  • Mitogens / pharmacology
  • Neoplastic Stem Cells / drug effects
  • Neoplastic Stem Cells / pathology
  • Okadaic Acid / pharmacology
  • Phytohemagglutinins / pharmacology
  • Sphingosine / analogs & derivatives*
  • Sphingosine / chemical synthesis
  • Sphingosine / pharmacology
  • T-Lymphocytes / drug effects*

Substances

  • Amino Acid Chloromethyl Ketones
  • Cysteine Proteinase Inhibitors
  • Mitogens
  • N-acetylsphingosine
  • Phytohemagglutinins
  • benzyloxycarbonylvalyl-alanyl-aspartyl fluoromethyl ketone
  • Okadaic Acid
  • Caspases
  • Sphingosine