The effect of staphylococcal enterotoxin B on thyrocyte HLA molecule expression

J Autoimmun. 1999 Jun;12(4):305-14. doi: 10.1006/jaut.1999.0279.

Abstract

Microbial superantigens have been implicated in the pathogenesis of human autoimmune diseases. In autoimmune glands, thyrocytes inappropriately express HLA-DR molecules and these cells may function as antigen presenting cells (APC) We studied the effect in vitro of staphylococcal enterotoxin B (SEB) on HLA molecule expression on thyrocytes obtained from autoimmune and non-autoimmune glands by immunofluorescence. HLA class I and class II upregulation could be detected by FACS analysis on thyrocytes. Anti-IFN-gamma neutralizing antibodies markedly affect both class I and class II upregulation on thyrocytes. FRTL5 cells were not responsive to SEB. Similarly, a human thyroid cell strain maintained in culture in a conditioned medium was not induced to express HLA products by SEB stimulation. The addition of autologous intrathyroidal lymphocytes caused reestablishment of the SEB effect.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cells, Cultured
  • Enterotoxins / pharmacology*
  • Flow Cytometry
  • Histocompatibility Antigens Class I / biosynthesis*
  • Histocompatibility Antigens Class II / biosynthesis*
  • Humans
  • Interferon-gamma / metabolism
  • Interferon-gamma / pharmacology
  • Staphylococcus aureus / immunology*
  • Superantigens / pharmacology*
  • Thyroid Gland / cytology
  • Thyroid Gland / drug effects*
  • Thyroid Gland / immunology
  • Up-Regulation

Substances

  • Enterotoxins
  • Histocompatibility Antigens Class I
  • Histocompatibility Antigens Class II
  • Superantigens
  • enterotoxin B, staphylococcal
  • Interferon-gamma