Abstract
Geldanamycin (GDM) binds to the Hsp90 chaperone protein and causes the degradation of several important signalling proteins. A series of novel estradiol-geldanamycin hybrids has been synthesized and evaluated for their ability to induce the selective degradation of the estrogen receptor (ER). The hybrid compounds are active and more selective than the parent causing degradation of ER and HER2, but not other GDM targets.
Publication types
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Research Support, U.S. Gov't, Non-P.H.S.
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Benzoquinones
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Enzyme Inhibitors / chemical synthesis*
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Estradiol / chemical synthesis*
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HSP90 Heat-Shock Proteins / antagonists & inhibitors
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Humans
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Inhibitory Concentration 50
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Lactams, Macrocyclic
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Models, Chemical
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Quinones / chemical synthesis*
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Receptors, Estrogen / antagonists & inhibitors
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Tumor Cells, Cultured
Substances
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Benzoquinones
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Enzyme Inhibitors
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HSP90 Heat-Shock Proteins
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Lactams, Macrocyclic
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Quinones
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Receptors, Estrogen
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Estradiol
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geldanamycin