Immune effector mechanisms in malaria

Curr Opin Immunol. 1999 Aug;11(4):412-9. doi: 10.1016/S0952-7915(99)80069-7.

Abstract

Malaria, a disease responsible for immense human suffering, is caused by infection with Plasmodium spp. parasites, which have a very complex life cycle - antigenically unique stages infect different tissues of the body. This review details recent developments in our understanding of immunity both to pre-erythrocytic stage antigens and to erythrocytic stage antigens. The former is largely mediated via CD8(+) T cells and involves IFN-gamma, nitric oxide, IL-12 and natural killer cells; the latter varies (in different hosts and with different parasites) but is largely mediated by antibody, helper T cells, nitric oxide and gammadelta T cells. The recent progress towards clinical trials of vaccine candidates against both the pre-erythrocytic stage and erythrocytic stage is also summarized, in particular the use of heterologous prime/boost strategies for the former and the use of MSP1 as a candidate vaccine for the latter.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Review

MeSH terms

  • Animals
  • CD4-Positive T-Lymphocytes / immunology
  • Erythrocytes / parasitology
  • Humans
  • Malaria / immunology*
  • Malaria Vaccines / immunology
  • Merozoite Surface Protein 1 / immunology
  • T-Lymphocytes, Cytotoxic / immunology

Substances

  • Malaria Vaccines
  • Merozoite Surface Protein 1