Abstract
The biological role(s) proposed for UCP3 in energy homeostasis have been based primarily upon amino acid sequence homology to UCP1. Spontaneous mutations of UCP3> have been described in humans, but not in rodents. The functional consequences-or lack thereof-of these mutations in humans will be of great importance in elucidating the biology of this protein. The results of two such studies are summarized here.
MeSH terms
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Animals
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Carrier Proteins / genetics
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Carrier Proteins / physiology*
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Energy Metabolism / genetics
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Energy Metabolism / physiology*
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Exons
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Homeostasis
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Humans
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Ion Channels
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Mitochondria / physiology*
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Mitochondrial Proteins
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Mutation
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Obesity / genetics
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Obesity / physiopathology
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Uncoupling Protein 3
Substances
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Carrier Proteins
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Ion Channels
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Mitochondrial Proteins
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UCP3 protein, human
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Uncoupling Protein 3