Axin directly interacts with plakoglobin and regulates its stability

J Biol Chem. 1999 Sep 24;274(39):27682-8. doi: 10.1074/jbc.274.39.27682.

Abstract

Plakoglobin is homologous to beta-catenin. Axin, a Wnt signal negative regulator, enhances glycogen synthase kinase (GSK)-3beta-dependent phosphorylation of beta-catenin and stimulates the degradation of beta-catenin. Therefore, we examined the effect of Axin on plakoglobin stability. Axin formed a complex with plakoglobin in COS cells and SW480 cells. Axin directly bound to plakoglobin, and this binding was inhibited by beta-catenin. Axin promoted GSK-3beta-dependent phosphorylation of plakoglobin. Furthermore, overexpression of Axin down-regulated the level of plakoglobin in SW480 cells. These results suggest that Axin regulates the stability of plakoglobin by enhancing its phosphorylation by GSK-3beta and that Axin may act on beta-catenin and plakoglobin in similar manners.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Axin Protein
  • COS Cells
  • Cell Line
  • Cytoskeletal Proteins / genetics
  • Cytoskeletal Proteins / isolation & purification
  • Cytoskeletal Proteins / metabolism*
  • Desmoplakins
  • Kinetics
  • Mitogen-Activated Protein Kinases / metabolism
  • Phosphorylation
  • Plasmids
  • Proteins / isolation & purification
  • Proteins / metabolism*
  • Recombinant Proteins / chemistry
  • Recombinant Proteins / metabolism
  • Repressor Proteins*
  • Trans-Activators*
  • Transfection
  • beta Catenin
  • gamma Catenin

Substances

  • Axin Protein
  • Cytoskeletal Proteins
  • Desmoplakins
  • Proteins
  • Recombinant Proteins
  • Repressor Proteins
  • Trans-Activators
  • beta Catenin
  • gamma Catenin
  • Mitogen-Activated Protein Kinases