Cooperative structural dynamics and a novel fidelity mechanism in histidyl-tRNA synthetases

Biochemistry. 1999 Sep 21;38(38):12296-304. doi: 10.1021/bi990482v.

Abstract

The crystal structure of the Staphylococcus aureus histidyl-tRNA synthetase apoprotein has been determined at 2.7 A resolution. Several important loops in the active site either become disordered or adopt very different conformations compared to their ligand-bound states. These include the histidine A motif (Arg257-Tyr262) that is essential for substrate recognition, a loop (Gly52-Lys62) that seems to control the communication between the histidine and ATP binding sites, the motif 2 loop (Glu114-Arg120) that binds ATP, and the insertion domain that is likely to bind tRNA. These ligand-induced structural changes are supported by fluorescence experiments, which also suggest highly cooperative dynamics. A dynamic and cooperative active site is most likely necessary for the proper functioning of the histidyl-tRNA synthetase, and suggests a novel mechanism for improving charging fidelity.

MeSH terms

  • Amino Acid Sequence
  • Binding Sites
  • Catalysis
  • Crystallography, X-Ray
  • Dimerization
  • Histidine / chemistry
  • Histidine / metabolism
  • Histidine-tRNA Ligase / chemistry*
  • Histidine-tRNA Ligase / metabolism*
  • Ligands
  • Models, Molecular
  • Molecular Sequence Data
  • Protein Conformation
  • Spectrometry, Fluorescence
  • Staphylococcus aureus / enzymology
  • Structure-Activity Relationship
  • Thermodynamics
  • Tryptophan / chemistry

Substances

  • Ligands
  • Histidine
  • Tryptophan
  • Histidine-tRNA Ligase

Associated data

  • PDB/1EQ0