Reduction in T cell apoptosis in patients with HIV disease following antiretroviral therapy

Clin Immunol. 1999 Oct;93(1):24-33. doi: 10.1006/clim.1999.4770.

Abstract

Patients with HIV infection manifest increased T lymphocyte apoptosis. This study investigated the influence of antiretroviral therapy (ART) upon lymphocyte apoptosis in 23 HIV-infected adults naive to protease inhibitors. Patients were enrolled in a treatment trial consisting of Nelfinavir (NFV), d4T, or NFV + d4T for 24 weeks, followed by triple therapy (NFV + reverse transcriptase inhibitors) for an additional 24 weeks. Spontaneous T cell apoptosis in cultured PBMC decreased by 23.67 +/- 18.2% (P < 0.006) at 48 weeks and plasma HIV RNA decreased by 1.79 +/- 0.59 log(10) RNA copies/ml (P < 0.001). The absolute decrease and slope of T cell apoptosis correlated with plasma virus load and with activated CD8 T cells and was inversely correlated with CD4 T cells. We conclude that reduction in chronic antigenic stimulation and the absence of cellular signals elicited by viral products contribute to the rescue of T lymphocytes from apoptosis, which facilitates immunologic recovery in ART-treated patients.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • Antiviral Agents / therapeutic use
  • Apoptosis
  • CD4 Lymphocyte Count
  • CD8-Positive T-Lymphocytes / cytology
  • Fas Ligand Protein
  • HIV Infections / pathology*
  • HIV Protease Inhibitors / therapeutic use
  • Humans
  • Lymphocyte Count
  • Membrane Glycoproteins / physiology
  • Nelfinavir / therapeutic use
  • RNA, Viral / blood
  • Retroviridae / drug effects*
  • Retroviridae / genetics
  • Signal Transduction
  • T-Lymphocytes / cytology*
  • Time Factors
  • Viral Load

Substances

  • Antiviral Agents
  • FASLG protein, human
  • Fas Ligand Protein
  • HIV Protease Inhibitors
  • Membrane Glycoproteins
  • RNA, Viral
  • Nelfinavir