The action of a novel vitamin D3 analogue, OCT, on immunomodulatory function of keratinocytes and lymphocytes

Arch Dermatol Res. 1999 Sep;291(9):500-6. doi: 10.1007/s004030050444.

Abstract

Topical vitamin D3 has relatively recently been introduced for the treatment of psoriasis. Synthetic vitamin D3 analogues with a high potential for inducing differentiation of cells, but with a low hypercalcemic effect have recently been developed. One such synthetic analogue of 1,25-dihydroxyvitamin D3 (calcitriol), 22-oxacalcitriol (OCT), is a novel agent for the topical treatment of psoriasis. The activity of OCT in vitro was investigated and compared with that of a series of vitamin D3 analogues as to their ability to inhibit murine T lymphocyte proliferation stimulated by con-A, to suppress IL-6 and IL-8 production by keratinocytes stimulated with IL-1alpha and TNFalpha, and to inhibit AP-1- and NFkappaB-dependent reporter gene expression. OCT inhibited the proliferation of lymphocytes and suppressed IL-8 and IL-6 production by keratinocytes to the same extent as the other vitamin D3 analogues. It also inhibited AP-1- and NFkappaB-controlled luciferase activity to the same extent as the other vitamin D3 analogues, which demonstrates its mechanism of action in the suppression of inflammatory processes.

MeSH terms

  • Animals
  • Calcitriol / analogs & derivatives*
  • Calcitriol / pharmacology
  • Cell Division / drug effects
  • Cholecalciferol / analogs & derivatives
  • Concanavalin A / pharmacology
  • Humans
  • Immune System / drug effects*
  • Interleukin-6 / antagonists & inhibitors
  • Interleukin-6 / biosynthesis
  • Interleukin-8 / antagonists & inhibitors
  • Interleukin-8 / biosynthesis
  • Jurkat Cells
  • Keratinocytes / drug effects*
  • Keratinocytes / immunology*
  • Keratinocytes / metabolism
  • Lymphocytes / cytology
  • Lymphocytes / drug effects*
  • Lymphocytes / immunology*
  • Mice
  • Mice, Inbred C3H
  • NF-kappa B / physiology
  • Transcription Factor AP-1 / physiology
  • Transcription, Genetic / drug effects
  • Transcription, Genetic / physiology

Substances

  • Interleukin-6
  • Interleukin-8
  • NF-kappa B
  • Transcription Factor AP-1
  • Concanavalin A
  • Cholecalciferol
  • Calcitriol
  • maxacalcitol