Present and potential future use of gene therapy for the treatment of non-insulin dependent diabetes mellitus (Review)

Int J Mol Med. 1999 Dec;4(6):585-92. doi: 10.3892/ijmm.4.6.585.

Abstract

This review describes the latest approaches towards using gene therapy as a treatment for non-insulin dependent diabetes mellitus (NIDDM; Type 2 diabetes). We examine attempts to directly deliver the insulin gene to non-beta-cells, to improve insulin secretion from existing beta-cells and to develop ex vivo approaches to implanting genetically modified cells. Future research into the pathology of non-insulin dependent diabetes, combined with the latest developments in gene delivery systems, may potentially make gene therapy an attractive alternative NIDDM treatment in the future.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Adult
  • Animals
  • Antigens, Neoplasm*
  • Biomarkers, Tumor*
  • Blood Glucose / analysis
  • Cell Transplantation
  • Diabetes Mellitus, Type 2 / genetics
  • Diabetes Mellitus, Type 2 / physiopathology
  • Diabetes Mellitus, Type 2 / therapy*
  • Gene Expression Regulation
  • Genes, Synthetic
  • Genetic Therapy*
  • Genetic Vectors
  • Glucagon / genetics
  • Glucagon / physiology
  • Glucagon-Like Peptide 1
  • Glucokinase / genetics
  • Glucokinase / physiology
  • Glucose Transporter Type 2
  • Homeodomain Proteins*
  • Humans
  • Hyperinsulinism / etiology
  • Hypoglycemic Agents / pharmacology
  • Hypoglycemic Agents / therapeutic use
  • Insulin / genetics*
  • Insulin / metabolism
  • Insulin Resistance
  • Insulin Secretion
  • Islets of Langerhans / drug effects
  • Islets of Langerhans / metabolism
  • Islets of Langerhans Transplantation
  • Lectins, C-Type*
  • Mice
  • Middle Aged
  • Monosaccharide Transport Proteins / genetics
  • Monosaccharide Transport Proteins / physiology
  • Muscle Contraction
  • Nitric Oxide / physiology
  • Nitric Oxide Synthase / genetics
  • Nitric Oxide Synthase / physiology
  • Nitric Oxide Synthase Type I
  • Pancreatitis-Associated Proteins
  • Peptide Fragments / genetics
  • Peptide Fragments / physiology
  • Promoter Regions, Genetic
  • Protein Precursors / genetics
  • Protein Precursors / physiology
  • Proteins / genetics
  • Proteins / physiology
  • Rats
  • Trans-Activators / genetics
  • Trans-Activators / physiology

Substances

  • Antigens, Neoplasm
  • Biomarkers, Tumor
  • Blood Glucose
  • Glucose Transporter Type 2
  • Homeodomain Proteins
  • Hypoglycemic Agents
  • Insulin
  • Lectins, C-Type
  • Monosaccharide Transport Proteins
  • Pancreatitis-Associated Proteins
  • Peptide Fragments
  • Protein Precursors
  • Proteins
  • REG3A protein, human
  • Reg3b protein, mouse
  • Trans-Activators
  • pancreatic and duodenal homeobox 1 protein
  • Nitric Oxide
  • Glucagon-Like Peptide 1
  • Glucagon
  • NOS1 protein, human
  • Nitric Oxide Synthase
  • Nitric Oxide Synthase Type I
  • Nos1 protein, mouse
  • Nos1 protein, rat
  • Glucokinase