Regulation of cutaneous allergic reaction by odorant inhalation

J Invest Dermatol. 2000 Mar;114(3):541-4. doi: 10.1046/j.1523-1747.2000.00896.x.

Abstract

Olfactory stimuli modulate emotional conditions and the whole body immune system. Effects of odorant inhalation on cutaneous immune reaction were examined. Contact hypersensitivity to 2,4, 6-trinitrochlorobenzene was elicited in C57BL/6 mice. The reaction was suppressed at both the induction and elicitation phases by exposure to an odorant, citralva. Topical application of citralva or lyral/lilial did not affect the reaction. The suppressive effect of citralva was more potent than that of another odorant, lyral/lilial. Citralva decreased the number of epidermal Langerhans cells, whereas lyral/lilial had a weak effect. Citralva but not lyral/lilial induced plasma corticosterone. Glucocorticoid receptor antagonist abrogated the suppressive effect of citralva on contact hypersensitivity. Serum interleukin-12 was downregulated by exposure to citralva or lyral/lilial. These data demonstrate that olfactory stimuli regulate the cutaneous immune system.

MeSH terms

  • Administration, Inhalation
  • Administration, Topical
  • Aldehydes / pharmacology
  • Allergens
  • Animals
  • Cyclohexenes
  • Cytokines / blood
  • Dermatitis, Contact / prevention & control*
  • Female
  • Interleukin-10 / blood
  • Interleukin-12 / blood
  • Langerhans Cells / drug effects
  • Mice
  • Mice, Inbred C57BL
  • Nitriles / pharmacology*
  • Odorants*
  • Perfume
  • Receptors, Odorant / pharmacology

Substances

  • Aldehydes
  • Allergens
  • Cyclohexenes
  • Cytokines
  • Nitriles
  • Perfume
  • Receptors, Odorant
  • Interleukin-10
  • Interleukin-12
  • citralva
  • hydroxyisohexyl 3-cyclohexene carboxaldehyde