Synaptic transmission induces transient Ca2+ concentration changes in cultured myenteric neurones

Neurogastroenterol Motil. 2000 Apr;12(2):117-24. doi: 10.1046/j.1365-2982.2000.00196.x.

Abstract

The enteric nervous system controls most of the gastrointestinal functions. We applied confocal microscopy and the Ca2+ indicator Fluo-3 as an optical approach to study synaptic activation in cultures of myenteric neurones. The optical recording of [Ca2+]i (the intracellular Ca2+ concentration) was used to monitor activation, since [Ca2+]i is crucial in the coupling between neuronal excitation and the activation of several intracellular events. Extracellular fibre tract stimulation (2 s, 30 Hz) caused a transient [Ca2+]i rise in a subset of neurones (50%). These transients lasted for 5.2 s (n=36), with an average amplitude of 3.4 +/- 1.3 times the basal concentration. The removal of extracellular Ca2+ (n=15) or the application of 10-6 M tetrodotoxin (n=16) blocked this response. The N-type Ca2+-channel blocker omega-conotoxin (5 x 10 -7M) abolished the [Ca2+]i increase, while blockade of L-type and P/Q type Ca2+ channels had no effect. Single stimuli evoked a [Ca2+]i rise in the processes. omega-conotoxin-sensitive postsynaptic events required repetitive stimulation. Cholinergic blockade did not inhibit the [Ca2+]i rise in all neurones, suggesting that, besides acetylcholine, other neurotransmitters are involved. Optical imaging of [Ca2+]i can be used to study synaptic spread of activation in enteric neuronal circuits expressed in culture.

Publication types

  • Comparative Study

MeSH terms

  • Action Potentials / drug effects
  • Animals
  • Atropine / pharmacology
  • Calcium Channel Blockers / classification
  • Calcium Channel Blockers / pharmacology
  • Calcium Signaling* / drug effects
  • Cells, Cultured / drug effects
  • Cholinergic Antagonists / pharmacology
  • Electric Stimulation
  • Excitatory Postsynaptic Potentials / drug effects
  • Guinea Pigs
  • Hexamethonium / pharmacology
  • Microscopy, Confocal
  • Myenteric Plexus / cytology
  • Myenteric Plexus / drug effects
  • Myenteric Plexus / physiology*
  • Nerve Net / drug effects
  • Nerve Net / physiology
  • Neurons / drug effects
  • Neurons / physiology*
  • Nicotinic Antagonists / pharmacology
  • Nifedipine / pharmacology
  • Parasympatholytics / pharmacology
  • Potassium / pharmacology
  • Receptors, Muscarinic / drug effects
  • Receptors, Muscarinic / physiology
  • Receptors, Nicotinic / drug effects
  • Receptors, Nicotinic / physiology
  • Synaptic Transmission / drug effects
  • Synaptic Transmission / physiology*
  • Tetrodotoxin / pharmacology
  • omega-Conotoxins / pharmacology

Substances

  • Calcium Channel Blockers
  • Cholinergic Antagonists
  • Nicotinic Antagonists
  • Parasympatholytics
  • Receptors, Muscarinic
  • Receptors, Nicotinic
  • omega-Conotoxins
  • Hexamethonium
  • Tetrodotoxin
  • Atropine
  • ziconotide
  • Nifedipine
  • Potassium