Inhibitory effect of cycloSaligenyl-nucleoside monophosphates (cycloSal-NMP) of acyclic nucleoside analogues on HSV-1 and EBV

Antiviral Res. 2000 Jan;45(1):69-77. doi: 10.1016/s0166-3542(99)00076-5.

Abstract

The in vitro antiviral activity of a new series of cycloSal-pro-nucleotides derived from the acyclic nucleoside analogues aciclovir and penciclovir against herpes simplex virus type 1 (HSV-1), thymidine kinase deficient (TK-) HSV-1, and Epstein-Barr virus (EBV) was evaluated. Using the XTT-based tetrazolium reduction assay EZ4U, the cycloSal derivatives were examined for their antiviral and cytotoxic effects in HSV-1 as well as HSV-1-TK--infected Vero cells. The anti-EBV activity was assessed by means of an EBV DNA hybridization assay using a digoxigenin-labeled probe specific for the Bam H1-W-fragment of the EBV genome and by measuring viral capsid antigen (VCA) expression in P3HR-1 cells by indirect immunofluorescence. Among the new cycloSal-phosphotriesters the three aciclovir monophosphates proved to be potent and selective inhibitors of HSV-1 replication, EBV DNA synthesis and EB-VCA expression. Of interest is the retention of activity of the aciclovir monophosphates in HSV-1-TK--infected cells. Particularly 3-methyl-cycloSal-aciclovir monophosphate retained the same effectiveness, as compared to the wild type virus strain. In contrast to the aciclovir pro-nucleotides the penciclovir cycloSal-phosphotriesters exhibited at best only a marginal antiviral effect on HSV and EBV replication.

MeSH terms

  • Acyclovir / analogs & derivatives*
  • Acyclovir / chemistry
  • Acyclovir / pharmacology
  • Animals
  • Antigens, Viral / biosynthesis
  • Antiviral Agents / chemistry
  • Antiviral Agents / pharmacology*
  • Capsid Proteins*
  • Cells, Cultured
  • Chlorocebus aethiops
  • DNA, Viral / biosynthesis
  • Guanine
  • Herpesvirus 1, Human / drug effects*
  • Herpesvirus 1, Human / metabolism
  • Herpesvirus 4, Human / drug effects*
  • Herpesvirus 4, Human / metabolism
  • Thymidine Kinase / metabolism
  • Vero Cells
  • Virus Replication / drug effects

Substances

  • Antigens, Viral
  • Antiviral Agents
  • Capsid Proteins
  • DNA, Viral
  • Epstein-Barr viral capsid antigen
  • penciclovir
  • Guanine
  • Thymidine Kinase
  • Acyclovir