Evidence for a parasite-mediated inhibition of abomasal acid secretion in sheep infected with Ostertagia leptospicularis

Vet J. 2000 May;159(3):238-51. doi: 10.1053/tvjl.1999.0475.

Abstract

The acid secretory capacity of the abomasal mucosa was studied in sheep experimentally infected with Ostertagia leptospicularis. The acidity of the abomasal contents, permanently recorded by a pH probe located inside the abomasum, decreased markedly to mean levels between pH 5 and 6. Subcutaneous administration of histamine or carbachol successfully stimulated acid secretion (pH 3.4). The results indicate that the abomasal mucosa harboured a population of functional parietal cells which were also identified immunohistochemically (H(+)/K(+)-ATPase). Ultrastructural investigation before stimulation revealed that the majority of these cells was in a resting state. Despite high serum gastrin levels, the acid secretion was blocked either at the level of the parietal cell or the enterochromaffin-like cell by an unknown factor, possibly mediated by the parasites. This is the first report of a parietal cell dysfunction associated with a nematode infection in the abomasum. It is suggested that the parasites induce changes in their environment which favour their survival and/or increase their reproduction.

MeSH terms

  • Abomasum / metabolism*
  • Abomasum / parasitology
  • Abomasum / ultrastructure
  • Animals
  • Feces / parasitology
  • Gastric Acid / metabolism*
  • Gastrins / blood
  • Gastroscopy / veterinary
  • Hydrogen-Ion Concentration
  • Immunohistochemistry
  • Microscopy, Electron / veterinary
  • Ostertagia / physiology*
  • Ostertagiasis / parasitology
  • Ostertagiasis / physiopathology
  • Ostertagiasis / veterinary*
  • Parietal Cells, Gastric / metabolism
  • Parietal Cells, Gastric / parasitology
  • Parietal Cells, Gastric / ultrastructure
  • Pepsinogen A / blood
  • Sheep
  • Sheep Diseases / parasitology*
  • Sheep Diseases / physiopathology*

Substances

  • Gastrins
  • Pepsinogen A