Potential mechanisms of human natural killer cell expansion in vivo during low-dose IL-2 therapy

J Clin Invest. 2000 Jul;106(1):117-24. doi: 10.1172/JCI6218.

Abstract

The continuous, in vivo infusion of low-dose IL-2 selectively expands the absolute number of human natural killer (NK) cells after 4-6 weeks of therapy. The mechanism responsible for this expansion is unknown and was examined in this study. NK cells cultured at low concentrations of IL-2, comparable to those found during in vivo therapy, proliferate for 6 days and then exit the cell cycle. However, NK cells in vivo did not traverse the S/G(2)/M phase of the cell cycle during low-dose IL-2 therapy. Low concentrations of IL-2 delay programmed cell death of NK cells but have the same effect on resting T cells that do not expand in vivo. When CD34(+) bone marrow hematopoietic progenitor cells are cultured for 21 days with low concentrations of IL-2, they differentiate into CD56(+)CD3(-) NK cells, not T cells. Thus, the selective expansion of human NK cells during continuous in vivo infusion of low-dose IL-2 likely results from enhanced NK-cell differentiation from bone marrow progenitors, combined with an IL-2-dependent delay in NK-cell death, rather than proliferation of mature NK cells in the periphery.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antigens, CD34 / analysis
  • CD56 Antigen / analysis
  • Humans
  • Interleukin-2 / pharmacology*
  • Interleukin-2 / therapeutic use
  • Killer Cells, Natural / drug effects*
  • Killer Cells, Natural / physiology
  • Lymphocyte Activation / drug effects
  • Rabbits
  • Recombinant Proteins / pharmacology

Substances

  • Antigens, CD34
  • CD56 Antigen
  • Interleukin-2
  • Recombinant Proteins