Peptide T does not ameliorate experimental autoimmune encephalomyelitis (EAE) in Lewis rats

Clin Exp Immunol. 2000 Jul;121(1):151-6. doi: 10.1046/j.1365-2249.2000.01259.x.

Abstract

Peptide T has been shown to inhibit T cell activation and cytokine production and function. Moreover, it has been reported to be a safe treatment in humans. We have studied the ability of peptide T to prevent or ameliorate EAE in Lewis rats. Peptide T was administered subcutaneously at different doses and phases of the disease according to several treatment protocols, but we could not observe a consistent effect of peptide T ameliorating the disease. Lymph node cell proliferation and IL-4 and interferon-gamma production were also studied. We conclude that peptide T neither prevents nor ameliorates EAE in Lewis rats.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Disease Models, Animal
  • Encephalomyelitis, Autoimmune, Experimental / drug therapy*
  • Encephalomyelitis, Autoimmune, Experimental / immunology
  • Encephalomyelitis, Autoimmune, Experimental / prevention & control
  • Female
  • Guinea Pigs
  • Interferon-gamma / biosynthesis
  • Interleukin-4 / biosynthesis
  • Peptide T / administration & dosage
  • Peptide T / therapeutic use*
  • Pilot Projects
  • Rats
  • Rats, Inbred Lew

Substances

  • Peptide T
  • Interleukin-4
  • Interferon-gamma