Extracellular ATP couples to cAMP generation and granulocytic differentiation in human NB4 promyelocytic leukaemia cells

Immunol Cell Biol. 2000 Oct;78(5):467-73. doi: 10.1111/j.1440-1711.2000.t01-4-.x.

Abstract

Priming of NB4 promyelocytic cells with all-trans retinoic acid, followed by extracellular ATP in the presence of a phosphodiesterase inhibitor, elevated cAMP and activated protein kinase A. The order of potency for cAMP production was ATP (EC50 = 95 +/- 13 micromol/L) > ADP > AMP = adenosine. The order of potency of ATP analogues was 2'- and 3'-O-(4-benzoylbenzoyl)-ATP (EC50 = 54 +/- 15 micromol/L) = adenosine 5'-O-(3-thio) triphosphate (EC50 = 66 +/- 4 micromol/L) > ATP > beta,gamma-methylene ATP (EC50 = 200 +/- 55 micromol/L). Adenosine 5'-O-thiomonophosphate and adenosine 5'-O-(2-thio) diphosphate inhibited ATP-induced cAMP production. Differentiation also occurred as measured by increased expression of CD11b and N-formyl peptide receptor and changes in cell morphology. UTP did not elevate cAMP or induce differentiation, indicating that P2Y2, P2Y4, and P2Y6 receptors were not involved. The P2Y11 receptor, a cAMP-linked receptor on promyelocytic HL-60 cells, was detected in NB4 cells by reverse transcription-polymerase chain reaction and northern blotting. This receptor has the same order of potency with respect to cAMP production as that observed in HL-60 cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 1-Methyl-3-isobutylxanthine / pharmacology
  • Adenine Nucleotides / metabolism
  • Adenosine Diphosphate / analogs & derivatives*
  • Adenosine Diphosphate / pharmacology
  • Adenosine Triphosphate / chemistry
  • Adenosine Triphosphate / metabolism*
  • Adenosine Triphosphate / pharmacology*
  • Antigens, CD / metabolism
  • Blotting, Northern
  • Cell Differentiation*
  • Cell Size / drug effects
  • Cyclic AMP / metabolism*
  • Cyclic AMP-Dependent Protein Kinases / metabolism
  • Dose-Response Relationship, Drug
  • Enzyme Activation
  • Granulocytes / cytology*
  • Granulocytes / drug effects
  • Granulocytes / metabolism
  • HL-60 Cells
  • Humans
  • Leukemia, Promyelocytic, Acute
  • N-Formylmethionine Leucyl-Phenylalanine / metabolism
  • Phosphodiesterase Inhibitors / pharmacology
  • Receptors, Formyl Peptide
  • Receptors, Immunologic / genetics
  • Receptors, Immunologic / metabolism
  • Receptors, Peptide / genetics
  • Receptors, Peptide / metabolism
  • Receptors, Purinergic P2 / genetics
  • Receptors, Purinergic P2 / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Thionucleotides / pharmacology
  • Tretinoin / pharmacology*
  • Tumor Cells, Cultured

Substances

  • Adenine Nucleotides
  • Antigens, CD
  • P2RY11 protein, human
  • Phosphodiesterase Inhibitors
  • Receptors, Formyl Peptide
  • Receptors, Immunologic
  • Receptors, Peptide
  • Receptors, Purinergic P2
  • Thionucleotides
  • adenosine 5'-O-(2-thiodiphosphate)
  • adenosine 5'-O-(1-thiodiphosphate)
  • Tretinoin
  • N-Formylmethionine Leucyl-Phenylalanine
  • Adenosine Diphosphate
  • Adenosine Triphosphate
  • Cyclic AMP
  • Cyclic AMP-Dependent Protein Kinases
  • 1-Methyl-3-isobutylxanthine