Lymphocytes lacking I kappa B-alpha develop normally, but have selective defects in proliferation and function

J Immunol. 2000 Nov 15;165(10):5418-27. doi: 10.4049/jimmunol.165.10.5418.

Abstract

NF-kappaB has been implicated in the development, activation, and function of B and T lymphocytes. We have evaluated the in vivo effects of deletion of IkappaB-alpha, a major inhibitor of NF-kappaB, on lymphocyte development, proliferation, and function. To elucidate the long term role of IkappaB-alpha in lymphocytes, fetal liver cells of 14.5-day-old IkappaB-alpha(-/-) or wild-type embryos were transplanted into irradiated recombinase-activating gene-2-deficient mice. Within 4 wk, the IkappaB-alpha(-/-) fetal liver cells reconstitute mature B and T cell populations in the recipients comparable to those produced by wild-type fetal liver cells. However, the proliferative responses of IkappaB-alpha(-/-) B cells are enhanced, whereas those of IkappaB-alpha(-/-) T cells are reduced. The levels of IgG1, IgG2a, IgA, and IgE produced by IkappaB-alpha(-/-) B cells are elevated relative to those produced by IkappaB-alpha(+/+) or IkappaB-alpha(+/-). Moreover, the specific immune responses to OVA and the generation of germinal centers are impaired in recipients of IkappaB-alpha(-/-) fetal liver cells. These results indicate that IkappaB-alpha plays a vital role in signal transduction pathways regulating lymphocyte proliferation and also in the production of specific Ig isotypes.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • B-Lymphocytes / immunology
  • B-Lymphocytes / metabolism
  • B-Lymphocytes / pathology
  • Cell Differentiation / genetics
  • Cell Differentiation / immunology
  • Cell Division / genetics
  • Cell Division / immunology
  • Clone Cells
  • DNA-Binding Proteins / deficiency*
  • DNA-Binding Proteins / genetics*
  • Epitopes, B-Lymphocyte / immunology
  • Fetal Tissue Transplantation / immunology
  • Fetal Tissue Transplantation / pathology
  • Fetus
  • Germinal Center / immunology
  • Germinal Center / pathology
  • I-kappa B Proteins*
  • Immunoglobulins / biosynthesis
  • Liver / cytology
  • Liver / embryology
  • Liver / growth & development
  • Liver / immunology
  • Liver Transplantation / immunology
  • Liver Transplantation / pathology
  • Lymphocyte Activation / genetics*
  • Lymphocyte Subsets / cytology
  • Lymphocyte Subsets / immunology*
  • Lymphocyte Subsets / metabolism*
  • Lymphocyte Subsets / pathology
  • Lymphoproliferative Disorders / genetics*
  • Lymphoproliferative Disorders / immunology
  • Lymphoproliferative Disorders / pathology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • NF-KappaB Inhibitor alpha
  • NF-kappa B / antagonists & inhibitors*
  • NF-kappa B / genetics
  • NF-kappa B / metabolism
  • Organ Specificity / genetics
  • Organ Specificity / immunology
  • Spleen / immunology
  • Spleen / pathology
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism
  • T-Lymphocytes / pathology
  • Transcriptional Activation / immunology

Substances

  • DNA-Binding Proteins
  • Epitopes, B-Lymphocyte
  • I-kappa B Proteins
  • Immunoglobulins
  • NF-kappa B
  • Nfkbia protein, mouse
  • NF-KappaB Inhibitor alpha