Clinical development of eniluracil/fluorouracil: an oral treatment for patients with solid tumors

Invest New Drugs. 2000 Nov;18(4):383-90. doi: 10.1023/a:1006457617467.

Abstract

Eniluracil (776C85, GW776) inactivates dihydropyrimidine dehydrogenase (DPD), the principal enzyme of 5-fluorouracil (5-FU) catabolism. Inactivation of DPD eliminates a potential mechanism for tumor 5-FU resistance and permits achievement of reliable and predictable pharmacokinetics following oral 5-FU administration. Eniluracil/5-FU has demonstrated efficacy as monotherapy in patients with a variety of solid tumors when given on a 5 or 28-day dosing schedule. The primary and dose-limiting toxicity is myelosuppression with the 5-day schedule and diarrhea with the 28-day schedule. The frequency of hand-foot syndrome is minimal with either schedule. Phase III pivotal registration-directed studies with eniluracil/5-FU given by the 28-day schedule are ongoing or planned for the near future in patients with advanced colorectal, breast and pancreatic cancer.

Publication types

  • Review

MeSH terms

  • Administration, Oral
  • Antimetabolites, Antineoplastic / administration & dosage
  • Antimetabolites, Antineoplastic / pharmacology
  • Antineoplastic Combined Chemotherapy Protocols / adverse effects
  • Antineoplastic Combined Chemotherapy Protocols / therapeutic use*
  • Clinical Trials as Topic
  • Drug Synergism
  • Enzyme Inhibitors / administration & dosage
  • Enzyme Inhibitors / pharmacology
  • Fluorouracil / administration & dosage
  • Fluorouracil / pharmacology
  • Humans
  • Neoplasms / drug therapy*
  • Uracil / administration & dosage
  • Uracil / analogs & derivatives*
  • Uracil / pharmacology

Substances

  • Antimetabolites, Antineoplastic
  • Enzyme Inhibitors
  • eniluracil
  • Uracil
  • Fluorouracil