15-Lipoxygenase-1 mediates nonsteroidal anti-inflammatory drug-induced apoptosis independently of cyclooxygenase-2 in colon cancer cells

Cancer Res. 2000 Dec 15;60(24):6846-50.

Abstract

We previously found (I. Shureiqi et al., Carcinogenesis (Lond.), 20: 1985-1995, 1999; I. Shureiqi et al, J. Natl. Cancer Inst., 92: 1136-1142, 2000) that (a) 15-lipoxygenase-1 (15-LOX-1) protein and its product 13-S-hydroxyoctadecadienoic acid (13-S-HODE) are decreased; and (b) nonsteroidal anti-inflammatory drug (NSAID)-induced 15-LOX-1 expression is critical to NSAID-induced apoptosis in colorectal cancer cells expressing cyclooxygenase-2 (COX-2). We used the NSAIDs sulindac sulfone (COX-2-independent) and NS-398 (a COX-2 inhibitor) to assess NSAID upregulation of 15-LOX-1 in relation to COX-2 inhibition during NSAID-induced apoptosis in the DLD-1 (COX-2-negative) colon cancer cell line. We found that: (a) NSAIDs up-regulated 15-LOX-1, which preceded apoptosis; and (b) 15-LOX-1 inhibition blocked NSAID-induced apoptosis, which was restored by 13-S-HODE but not by its parent, linoleic acid. NSAIDs can induce apoptosis in colon cancer cells via up-regulation of 15-LOX-1 in the absence of COX-2.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Anti-Inflammatory Agents, Non-Steroidal / pharmacology*
  • Antineoplastic Agents / pharmacology
  • Antioxidants / pharmacology
  • Antithrombins / pharmacology
  • Apoptosis / drug effects*
  • Arachidonate 15-Lipoxygenase / pharmacology*
  • Arachidonic Acid / pharmacology
  • Blotting, Western
  • Caffeic Acids / pharmacology
  • Cell Line
  • Colonic Neoplasms / enzymology*
  • Colonic Neoplasms / pathology*
  • Cyclooxygenase 1
  • Cyclooxygenase 2
  • Cyclooxygenase 2 Inhibitors
  • Cyclooxygenase Inhibitors / pharmacology
  • Humans
  • Hydroxyeicosatetraenoic Acids / biosynthesis
  • Isoenzymes / metabolism*
  • Linoleic Acid / pharmacology
  • Linoleic Acids / pharmacology
  • Membrane Proteins
  • Nitrobenzenes / pharmacology
  • Prostaglandin-Endoperoxide Synthases / metabolism*
  • Recombinant Proteins / metabolism
  • Sulfonamides / pharmacology
  • Sulindac / analogs & derivatives
  • Sulindac / pharmacology
  • Time Factors
  • Tumor Cells, Cultured
  • Up-Regulation

Substances

  • Anti-Inflammatory Agents, Non-Steroidal
  • Antineoplastic Agents
  • Antioxidants
  • Antithrombins
  • Caffeic Acids
  • Cyclooxygenase 2 Inhibitors
  • Cyclooxygenase Inhibitors
  • Hydroxyeicosatetraenoic Acids
  • Isoenzymes
  • Linoleic Acids
  • Membrane Proteins
  • Nitrobenzenes
  • Recombinant Proteins
  • Sulfonamides
  • N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide
  • Sulindac
  • Arachidonic Acid
  • 13-hydroxy-9,11-octadecadienoic acid
  • Linoleic Acid
  • Arachidonate 15-Lipoxygenase
  • Cyclooxygenase 1
  • Cyclooxygenase 2
  • PTGS1 protein, human
  • PTGS2 protein, human
  • Prostaglandin-Endoperoxide Synthases
  • sulindac sulfone
  • caffeic acid