Targeting of HIV-1 Nef to the centrosome: implications for antigen processing

Traffic. 2000 Nov;1(11):884-91. doi: 10.1034/j.1600-0854.2000.011107.x.

Abstract

To gain a better understanding of the intracellular sites of antigen processing we have looked at the localization of human immunodeficiency virus (HIV)-1 Nef protein by confocal microscopic and biochemical means. We found that ubiquitin (Ub)-Nef fusion proteins were localized to the centrosome in transfected COS-7 cells, and that the colocalization was inhibited by the microtubule-disrupting agent, nocodazole. Interestingly, we found that Ub-Nef trafficking to the centrosome was not dependent upon the metabolic stability of Ub-Nef nor on the inhibition of proteasome activity. We also analyzed the MHC class I antigen processing of a reporter epitope linked to the Ub-Nef fusion proteins and found that Ub-Nef was processed in COS-7 cells. In addition, we show that this processing was inhibited by nocodazole. We suggest that the centrosome may serve as a site of antigen processing in vivo.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antigen Presentation / drug effects
  • Base Sequence
  • COS Cells
  • Centrosome / drug effects
  • Centrosome / immunology
  • Centrosome / metabolism*
  • DNA Primers / genetics
  • Gene Products, nef / genetics
  • Gene Products, nef / immunology
  • Gene Products, nef / metabolism*
  • HIV-1 / genetics
  • HIV-1 / immunology
  • HIV-1 / metabolism*
  • Humans
  • Nocodazole / pharmacology
  • Recombinant Fusion Proteins / genetics
  • Recombinant Fusion Proteins / metabolism
  • nef Gene Products, Human Immunodeficiency Virus

Substances

  • DNA Primers
  • Gene Products, nef
  • Recombinant Fusion Proteins
  • nef Gene Products, Human Immunodeficiency Virus
  • Nocodazole