Oxidative post-translational modifications of alpha-synuclein in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) mouse model of Parkinson's disease

J Neurochem. 2001 Jan;76(2):637-40. doi: 10.1046/j.1471-4159.2001.00174.x.

Abstract

Structural and functional alterations of alpha-synuclein is a presumed culprit in the demise of dopaminergic neurons in Parkinson's disease (PD). Alpha-synuclein mutations are found in familial but not in sporadic PD, raising the hypothesis that effects similar to those of familial PD-linked alpha-synuclein mutations may be achieved by oxidative post-translational modifications. Here, we show that wild-type alpha-synuclein is a selective target for nitration following peroxynitrite exposure of stably transfected HEK293 cells. Nitration of alpha-synuclein also occurs in the mouse striatum and ventral midbrain following administration of the parkinsonian neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP). Conversely, beta-synuclein and synaptophysin were not nitrated in MPTP-intoxicated mice. Our data demonstrate that alpha-synuclein is a target for tyrosine nitration, which, by disrupting its biophysical properties, may be relevant to the putative role of alpha-synuclein in the neurodegeneration associated with MPTP toxicity and with PD.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine*
  • Animals
  • Cell Line
  • Corpus Striatum / chemistry
  • Corpus Striatum / drug effects
  • Corpus Striatum / metabolism
  • Disease Models, Animal
  • Humans
  • Kidney / chemistry
  • Kidney / cytology
  • Kidney / drug effects
  • Kidney / metabolism
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Nerve Tissue Proteins / chemistry
  • Nerve Tissue Proteins / genetics
  • Nerve Tissue Proteins / metabolism*
  • Oxidation-Reduction / drug effects
  • Parkinsonian Disorders / chemically induced
  • Parkinsonian Disorders / metabolism*
  • Peroxynitrous Acid / pharmacology
  • Precipitin Tests
  • Protein Processing, Post-Translational / physiology*
  • Synaptophysin / analysis
  • Synaptophysin / metabolism
  • Synucleins
  • Transfection
  • Tyrosine / chemistry
  • Tyrosine / drug effects
  • Tyrosine / metabolism
  • alpha-Synuclein
  • beta-Synuclein

Substances

  • Nerve Tissue Proteins
  • SNCA protein, human
  • SNCB protein, human
  • Snca protein, mouse
  • Sncb protein, mouse
  • Synaptophysin
  • Synucleins
  • alpha-Synuclein
  • beta-Synuclein
  • Peroxynitrous Acid
  • Tyrosine
  • 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine