HLA-associated cellular response to GAD in type 2 diabetes with antibodies to GAD

Endocr J. 2000 Dec;47(6):753-61. doi: 10.1507/endocrj.47.753.

Abstract

Proliferative response of peripheral blood mononuclear cells (PBMC) to glutamic acid decarboxylase (GAD), which has been reported in patients with type 1 diabetes, was measured in type 2 diabetes, especially in patients with antibodies to GAD initially diagnosed as having type 2 diabetes (anti-GAD+ type 2 diabetes). We studied 12 patients with type 1 diabetes, 22 with anti-GAD+ type 2 diabetes, 31 with type 2 diabetes who were negative for anti-GAD (anti-GAD+ type 2 diabetes), and 30 healthy control subjects for cellular responses in vitro to GAD. The mean stimulation index (SI) in response to GAD was significantly higher in type 1 diabetes than in anti-GAD+ type 2 diabetes or healthy controls (1.47+/-0.35 vs. 1.11+/-0.35, P<0.05, and 1.06+/-0.07, P<0.05, respectively). The mean

MeSH terms

  • Adult
  • Aged
  • Alleles
  • Autoantibodies / blood*
  • Autoantigens / immunology
  • Cell Division / drug effects
  • Diabetes Mellitus, Type 1 / genetics
  • Diabetes Mellitus, Type 1 / immunology
  • Diabetes Mellitus, Type 2 / genetics*
  • Diabetes Mellitus, Type 2 / immunology*
  • Female
  • Genetic Predisposition to Disease
  • Glutamate Decarboxylase / immunology*
  • HLA Antigens / genetics*
  • HLA-DR Antigens / genetics
  • HLA-DRB1 Chains
  • Humans
  • Insulin / pharmacology
  • Male
  • Middle Aged
  • Polymerase Chain Reaction

Substances

  • Autoantibodies
  • Autoantigens
  • HLA Antigens
  • HLA-DR Antigens
  • HLA-DRB1 Chains
  • Insulin
  • Glutamate Decarboxylase