Temporal effects of inactivty on myosin heavy chain gene expression in rat slow muscle

Muscle Nerve. 2001 Apr;24(4):517-26. doi: 10.1002/mus.1035.

Abstract

Myosin heavy chain (MHC) mRNA and protein profiles in adult rat soleus and adductor longus were determined after 4, 8, 15, 30, 60, and 90 days of spinal cord isolation (SI). SI results in complete neuromuscular inactivity while leaving the motoneuron-muscle fiber connections intact. From 15 to 90 days, type I MHC mRNA was significantly decreased, whereas type I MHC protein did not significantly decrease until 30 and 60 days in the soleus and adductor longus, respectively. However, in both muscles, slow MHC downregulation was offset by significant upregulation of the faster MHC isoforms, primarily IIx. From 60 to 90 days, type I MHC was almost completely replaced with faster isoforms at the mRNA and protein levels. Thus, chronic inactivity and unloading of slow rat hindlimb muscles shifted the MHC profile from predominately type I to type IIx MHC mRNA and protein.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Body Weight
  • Disease Models, Animal
  • Female
  • Gene Expression*
  • Motor Activity / genetics*
  • Muscle Fibers, Slow-Twitch / metabolism*
  • Muscle Fibers, Slow-Twitch / pathology
  • Muscle, Skeletal / chemistry
  • Muscle, Skeletal / metabolism*
  • Muscle, Skeletal / pathology
  • Muscle, Skeletal / physiopathology
  • Myosin Heavy Chains / biosynthesis*
  • Myosin Heavy Chains / genetics
  • Organ Size
  • Protein Isoforms / biosynthesis
  • Protein Isoforms / genetics
  • RNA, Messenger / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Spinal Cord Injuries / metabolism
  • Spinal Cord Injuries / physiopathology
  • Time Factors

Substances

  • Protein Isoforms
  • RNA, Messenger
  • Myosin Heavy Chains