Bisphosphonates inhibit the growth of Trypanosoma brucei, Trypanosoma cruzi, Leishmania donovani, Toxoplasma gondii, and Plasmodium falciparum: a potential route to chemotherapy

J Med Chem. 2001 Mar 15;44(6):909-16. doi: 10.1021/jm0002578.

Abstract

We have investigated the effects in vitro of a series of bisphosphonates on the proliferation of Trypanosoma cruzi, Trypanosoma brucei rhodesiense, Leishmania donovani, Toxoplasma gondii, and Plasmodium falciparum. The results show that nitrogen-containing bisphosphonates of the type used in bone resorption therapy have significant activity against parasites, with the aromatic species having in some cases nanomolar or low-micromolar IC(50) activity values against parasite replication (e.g. o-risedronate, IC(50) = 220 nM for T. brucei rhodesiense; risedronate, IC(50) = 490 nM for T. gondii). In T. cruzi, the nitrogen-containing bisphosphonate risedronate is shown to inhibit sterol biosynthesis at a pre-squalene level, most likely by inhibiting farnesylpyrophosphate synthase. Bisphosphonates therefore appear to have potential in treating parasitic protozoan diseases.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antiprotozoal Agents / pharmacology*
  • Chlorocebus aethiops
  • Diphosphonates / pharmacology*
  • Leishmania donovani / drug effects
  • Plasmodium falciparum / drug effects
  • Structure-Activity Relationship
  • Toxoplasma / drug effects
  • Trypanosoma brucei brucei / drug effects
  • Trypanosoma cruzi / drug effects
  • Vero Cells

Substances

  • Antiprotozoal Agents
  • Diphosphonates