Immune evasion mechanisms of varicella-zoster virus

Arch Virol Suppl. 2001:(17):99-107. doi: 10.1007/978-3-7091-6259-0_11.

Abstract

Varicella-zoster virus can to modulate the expression of class I and class II major histocompatibility (MHC) molecules. MHC class I expression is downregulated in VZV-infected T cells as well as in fibroblasts. VZV-infected cells do not respond to exposure to interferon-gamma (IFN-gamma) by upregulation of MHC class II expression. However, MHC class II expression is induced when cells are treated with IFN-gamma before VZV infection. These effects on MHC class I and class II expression can be expected to interfere transiently with adaptive immune responses of the host, mediated by CD4 and CD8 T cells, ensuring that the virus has sufficient opportunity for transmission to susceptible contracts.

Publication types

  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Animals
  • Down-Regulation / immunology*
  • Herpesvirus 3, Human / immunology*
  • Herpesvirus 3, Human / physiology
  • Histocompatibility Antigens Class I / biosynthesis*
  • Histocompatibility Antigens Class II / biosynthesis*
  • Humans
  • Interferon-gamma / immunology
  • Up-Regulation / immunology*

Substances

  • Histocompatibility Antigens Class I
  • Histocompatibility Antigens Class II
  • Interferon-gamma