Survival and plasticity of basal forebrain cholinergic systems in mice transgenic for presenilin-1 and amyloid precursor protein mutant genes

Neuroreport. 2001 May 25;12(7):1377-84. doi: 10.1097/00001756-200105250-00018.

Abstract

The basalo-cortical cholinergic system was characterized in mice expressing mutant human genes for presenilin-1 (PS1), amyloid precursor protein (APP), and combined PS/APP. Dual immunocytochemistry for ChAT and A beta revealed swollen cholinergic processes within cortical plaques in both APP and PS/APP brains by 12 months, suggesting aberrant sprouting or redistribution of cholinergic processes in response to amyloid deposition. At 8 months, cortical and subcortical ChAT activity was normal (PS/APP) or elevated (PS, APP frontal cortex), while cholinergic cell counts (nBM/SI) and receptor binding were unchanged. ChAT mRNA was up-regulated in the nBM/SI of all three transgenic lines at 8 months. The data indicate that the basal forebrain cholinergic system does not degenerate in mice expressing AD-related transgenes, even in mice with extreme amyloid load. The

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acetylcholine / metabolism
  • Aging / physiology
  • Alzheimer Disease / genetics
  • Alzheimer Disease / metabolism
  • Alzheimer Disease / physiopathology
  • Amyloid beta-Protein Precursor / genetics*
  • Amyloid beta-Protein Precursor / metabolism
  • Animals
  • Basal Nucleus of Meynert / enzymology
  • Basal Nucleus of Meynert / growth & development
  • Basal Nucleus of Meynert / pathology*
  • Cell Count
  • Cell Survival / genetics*
  • Cerebral Cortex / enzymology
  • Cerebral Cortex / growth & development
  • Cerebral Cortex / pathology*
  • Choline O-Acetyltransferase / genetics
  • Choline O-Acetyltransferase / metabolism
  • Cholinergic Fibers / metabolism
  • Cholinergic Fibers / pathology*
  • Immunohistochemistry
  • Membrane Proteins / genetics*
  • Membrane Proteins / metabolism
  • Mice
  • Mice, Transgenic / abnormalities
  • Mice, Transgenic / metabolism
  • Nerve Degeneration / genetics
  • Nerve Degeneration / metabolism
  • Nerve Degeneration / physiopathology
  • Neuronal Plasticity / genetics*
  • Plaque, Amyloid / genetics
  • Plaque, Amyloid / metabolism
  • Plaque, Amyloid / pathology
  • Presenilin-1
  • RNA, Messenger / metabolism
  • Radioligand Assay
  • Receptors, Muscarinic / drug effects
  • Receptors, Muscarinic / metabolism
  • Receptors, Nicotinic / drug effects
  • Receptors, Nicotinic / metabolism
  • Up-Regulation / genetics

Substances

  • Amyloid beta-Protein Precursor
  • Membrane Proteins
  • PSEN1 protein, human
  • Presenilin-1
  • RNA, Messenger
  • Receptors, Muscarinic
  • Receptors, Nicotinic
  • Choline O-Acetyltransferase
  • Acetylcholine