Abstract
Transcription factor NF-kappaB is biochemically coupled to the T cell antigen receptor (TCR) and activated transiently during an adaptive immune response. The author's laboratory is investigating the signal-dependent regulation of NF-kappaB, its downstream gene targets, and its function in lymphocyte biology. Our studies have revealed novel enzymatic checkpoints in the NF-kappaB signaling pathway and constitutive repressors of NF-kappaB that might be clinically applicable for therapeutic control of the immune system. We have also found that the Tax transforming protein encoded by human T cell leukemia virus type 1 (HTLV1) binds to and persistently activates an inducible protein kinase in the TCR/NF-kappaB axis. This viral/host interaction appears to trigger the inappropriate expression of NF-kappaB and the development of HTLV1-associated disease.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, P.H.S.
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Review
MeSH terms
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Active Transport, Cell Nucleus
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Animals
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Cell Cycle
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DNA-Binding Proteins / chemistry
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DNA-Binding Proteins / genetics
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DNA-Binding Proteins / physiology
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Enzyme Induction
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Gene Expression Regulation
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Gene Products, tax / physiology
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HIV / physiology
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HTLV-I Infections / immunology
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Human T-lymphotropic virus 1 / physiology
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Humans
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I-kappa B Proteins*
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Interleukin-2 / biosynthesis
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Interleukin-2 / genetics
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Lymphocyte Activation / physiology*
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Macromolecular Substances
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Mice
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NF-KappaB Inhibitor alpha
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NF-kappa B / antagonists & inhibitors
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NF-kappa B / physiology*
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Protein Engineering
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Receptors, Interleukin-2 / biosynthesis
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Receptors, Interleukin-2 / genetics
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Signal Transduction / physiology*
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T-Lymphocytes / cytology
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T-Lymphocytes / immunology*
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T-Lymphocytes / metabolism
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Transcription, Genetic
Substances
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DNA-Binding Proteins
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Gene Products, tax
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I kappa B beta protein
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I-kappa B Proteins
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Interleukin-2
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Macromolecular Substances
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NF-kappa B
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NFKBIA protein, human
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Nfkbia protein, mouse
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Receptors, Interleukin-2
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NF-KappaB Inhibitor alpha