Frasier syndrome with childhood-onset renal failure

Horm Res. 2001;55(2):77-80. doi: 10.1159/000049974.

Abstract

Background: The Wilms' tumour 1 (WT1) gene encodes a protein which is believed to exert transcriptional and tumour-suppressor activities. Mutations of this gene have occasionally been associated with Wilms' tumour (<15% of cases) and, more consistently, with three syndromes characterized by urogenital abnormalities (WAGR, Denys-Drash and Frasier syndrome). SUBJECT/METHOD: A 25-year-old phenotypic female with a 46,XY karyotype presented with amenorrhoea. An ultrasound scan showed streak gonads and a rudimentary uterus. The patient had a history of post-streptococcal glomerulonephrosis, when aged 4 years, which had rapidly progressed to kidney failure, requiring transplantation at age 8.

Result: Frasier syndrome was suspected and confirmed by genetic analysis. In fact, direct sequencing of the PCR product of the intron 9 donor splice site revealed a substitution of guanine for adenine in position +5.

Conclusion: Besides being one of the few Frasier syndrome cases to be genetically characterized, this case is interesting because of the unusually early-onset renal failure.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Alternative Splicing
  • Disorders of Sex Development / genetics
  • Genes, Wilms Tumor / genetics*
  • Glomerulonephritis / complications
  • Glomerulonephritis / microbiology
  • Humans
  • Karyotyping
  • Kidney Transplantation
  • Male
  • Mutation
  • Phenotype
  • Renal Insufficiency / genetics*
  • Renal Insufficiency / surgery
  • Streptococcal Infections
  • Syndrome