Meiotic recombination, cross-reactivity, and persistence in Plasmodium falciparum

Evolution. 2001 Jul;55(7):1299-307. doi: 10.1111/j.0014-3820.2001.tb00652.x.

Abstract

We incorporate a representation of Plasmodium falciparum recombination within a discrete-event model of malaria transmission. We simulate the introduction of a new parasite genotype into a human population in which another genotype has reached equilibrium prevalence and compare the emergence and persistence of the novel recombinant forms under differing cross-reactivity relationships between the genotypes. Cross-reactivity between the parental (initial and introduced) genotypes reduces the frequency of appearance of recombinants within three years of introduction from 100% to 14%, and delays their appearance by more than a year, on average. Cross-reactivity between parental and recombinant genotypes reduces the frequency of appearance to 36% and increases the probability of recombinant extinction following appearance from 0% to 83%. When a recombinant is cross-reactive with its parental types, its probability of extinction is influenced by cross-reactivity between the parental types in the opposite manner; that is, its probability of extinction after appearance decreases. Frequencies of P. falciparum outcrossing are mediated by frequencies of mixed-genotype infections in the host population, which are in turn mediated by the structure of cross-reactivity between parasite genotypes. The three leading hypotheses about how meiosis relates to oocyst production lead to quantitative, but no qualitative, differences in these results.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Alleles
  • Animals
  • Cross Reactions*
  • Culicidae / parasitology
  • Evolution, Molecular*
  • Female
  • Genotype
  • Humans
  • Malaria, Falciparum / immunology*
  • Malaria, Falciparum / parasitology*
  • Malaria, Falciparum / transmission
  • Meiosis / genetics*
  • Models, Biological
  • Plasmodium falciparum / genetics*
  • Plasmodium falciparum / immunology*
  • Plasmodium falciparum / pathogenicity
  • Plasmodium falciparum / physiology
  • Recombination, Genetic*
  • Virulence