EBNA2 and Notch signalling in Epstein-Barr virus mediated immortalization of B lymphocytes

Semin Cancer Biol. 2001 Dec;11(6):423-34. doi: 10.1006/scbi.2001.0409.

Abstract

Epstein-Barr virus (EBV) has the ability to immortalize B cells. A viral key protein for immortalization is the transactivator EBNA2 that controls expression of several viral and cellular genes. EBNA2 is tethered to promoters by interacting with the cellular repressor RBP-J. This resembles the physiological activation of RBP-J-repressed promoters by activated Notch receptors (Notch-IC). Since EBNA2 and Notch-IC have been shown to be partially interchangeable in regard to activation of target genes in B cell lines and modulation of differentiation processes it is conceivable that EBNA2 is a biological equivalent of an activated Notch receptor.

Publication types

  • Review

MeSH terms

  • B-Lymphocytes / cytology*
  • B-Lymphocytes / virology*
  • Cell Survival
  • Cell Transformation, Viral
  • DNA-Binding Proteins / metabolism
  • Epstein-Barr Virus Nuclear Antigens / chemistry
  • Epstein-Barr Virus Nuclear Antigens / genetics
  • Epstein-Barr Virus Nuclear Antigens / metabolism*
  • Herpesvirus 4, Human / genetics
  • Herpesvirus 4, Human / metabolism*
  • Humans
  • Immunoglobulin J Recombination Signal Sequence-Binding Protein
  • Membrane Proteins / chemistry
  • Membrane Proteins / metabolism*
  • Nuclear Proteins*
  • Receptors, Notch
  • Signal Transduction*
  • Transcriptional Activation
  • Viral Proteins

Substances

  • DNA-Binding Proteins
  • EBNA-2 protein, Human herpesvirus 4
  • Epstein-Barr Virus Nuclear Antigens
  • Immunoglobulin J Recombination Signal Sequence-Binding Protein
  • Membrane Proteins
  • Nuclear Proteins
  • RBPJ protein, human
  • Receptors, Notch
  • Viral Proteins