Abstract
B lymphocyte development is regulated at multiple checkpoints, mediated by signals originating both inside and outside the cell. Two signaling pathways known to be essential in this process are interleukin-7 (IL-7) and the pre-B cell receptor (pBCR). We have shown previously that these signaling pathways intersect functionally. Specifically, response to low concentrations of IL-7 requires pBCR expression. In this report, we identify the ERK/MAP kinase pathway as a key regulatory component of this response. We propose a molecular mechanism for the selective expansion of pBCR(+) precursors and for the culling of inappropriately rearranged pro-B cells.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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B-Lymphocytes / cytology
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B-Lymphocytes / drug effects
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B-Lymphocytes / immunology*
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Bone Marrow / embryology
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Cell Differentiation
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Cell Division / drug effects
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Cell Lineage
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Cell Survival
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Cells, Cultured
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Dose-Response Relationship, Drug
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Hematopoietic Stem Cells / drug effects
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Hematopoietic Stem Cells / immunology
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Interleukin-7 / pharmacology*
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MAP Kinase Signaling System*
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Mice
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Mitogen-Activated Protein Kinases / physiology*
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Receptors, Antigen, B-Cell / metabolism*
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Receptors, Interleukin-7 / metabolism
Substances
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Interleukin-7
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Receptors, Antigen, B-Cell
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Receptors, Interleukin-7
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Mitogen-Activated Protein Kinases