Lymphotoxins and cytomegalovirus cooperatively induce interferon-beta, establishing host-virus détente

Immunity. 2001 Oct;15(4):617-26. doi: 10.1016/s1074-7613(01)00222-9.

Abstract

Tumor necrosis factor (TNF)-related cytokines regulate cell death and survival and provide strong selective pressures for viruses, such as cytomegalovirus (CMV), to evolve counterstrategies in order to persist in immune-competent hosts. Signaling by the lymphotoxin (LT)-beta receptor or TNF receptor-1, but not Fas or TRAIL receptors, inhibits the cytopathicity and replication of human CMV by a nonapoptotic, reversible process that requires nuclear factor kappa B (NF-kappa B)-dependent induction of interferon-beta (IFN-beta). Efficient induction of IFN-beta requires virus infection and LT signaling, demonstrating the need for both host and viral factors in the curtailment of viral replication without cellular elimination. LT alpha-deficient mice and LT beta R-Fc transgenic mice were profoundly susceptible to murine CMV infection. Together, these results reveal an essential and conserved role for LTs in establishing host defense to CMV.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adaptor Proteins, Signal Transducing*
  • Animals
  • Carrier Proteins / physiology
  • Cells, Cultured
  • Cytomegalovirus / growth & development
  • Cytomegalovirus / pathogenicity
  • Cytomegalovirus / physiology*
  • Fas-Associated Death Domain Protein
  • Herpesviridae Infections / etiology
  • Host-Parasite Interactions*
  • Humans
  • Interferon-beta / biosynthesis*
  • Interferon-beta / genetics
  • Interferon-beta / physiology
  • Lymphotoxin beta Receptor
  • Lymphotoxin-alpha / genetics
  • Lymphotoxin-alpha / pharmacology*
  • Membrane Proteins / pharmacology*
  • Mice
  • Mice, Transgenic
  • Muromegalovirus
  • NF-kappa B / physiology
  • Proteins / physiology
  • RNA, Messenger / biosynthesis
  • Receptors, Tumor Necrosis Factor / genetics
  • Survival Rate
  • TNF Receptor-Associated Factor 3
  • Transcriptional Activation*
  • Tumor Necrosis Factor Ligand Superfamily Member 14
  • Tumor Necrosis Factor-alpha / pharmacology*
  • Virus Replication / drug effects

Substances

  • Adaptor Proteins, Signal Transducing
  • Carrier Proteins
  • FADD protein, human
  • Fadd protein, mouse
  • Fas-Associated Death Domain Protein
  • LTBR protein, human
  • Ltbr protein, mouse
  • Lymphotoxin beta Receptor
  • Lymphotoxin-alpha
  • Membrane Proteins
  • NF-kappa B
  • Proteins
  • RNA, Messenger
  • Receptors, Tumor Necrosis Factor
  • TNF Receptor-Associated Factor 3
  • TNFSF14 protein, human
  • Tnfsf14 protein, mouse
  • Tumor Necrosis Factor Ligand Superfamily Member 14
  • Tumor Necrosis Factor-alpha
  • Interferon-beta