Molecular and cellular permeability control at the blood-brain barrier

Brain Res Brain Res Rev. 2001 Oct;36(2-3):258-64. doi: 10.1016/s0165-0173(01)00102-3.

Abstract

The blood-brain barrier (BBB) is formed by brain capillary endothelial cells. These cells have at least three properties which distinguish them from their peripheral counterparts: (1) tight junctions (TJs) of extremely low permeability; (2) low rates of fluid-phase endocytosis; (3) specific transport and carrier molecules. In combination, these features restrict the nonspecific flux of ions, proteins, and other substances into the central nervous system (CNS) environment. The restriction protects neurons from harmful compositional fluctuations occurring in the blood and allows uptake of essential molecules. Breakdown of the BBB is associated with a variety of CNS disorders and results in aggravation of the condition. Restoration of the BBB is thus one strategy during therapy of CNS diseases. Its success depends on a precise knowledge of the structural and functional principles underlying BBB functionality. In this review we have tried to summarise the current knowledge of TJs, including information gained from non-neuronal systems, and describe selected mechanisms involved in permeability regulation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Blood-Brain Barrier / drug effects
  • Blood-Brain Barrier / immunology*
  • Cell Membrane Permeability / drug effects
  • Cell Membrane Permeability / immunology*
  • Cell Movement / drug effects
  • Cell Movement / immunology
  • Central Nervous System Diseases / drug therapy*
  • Central Nervous System Diseases / immunology
  • Central Nervous System Diseases / metabolism
  • Cytokines / immunology
  • Cytokines / metabolism
  • Endothelium, Vascular / drug effects
  • Endothelium, Vascular / immunology
  • Endothelium, Vascular / metabolism*
  • Humans
  • Tight Junctions / drug effects
  • Tight Junctions / immunology
  • Tight Junctions / metabolism*

Substances

  • Cytokines