Laminin-1-induced migration of multiple myeloma cells involves the high-affinity 67 kD laminin receptor

Br J Cancer. 2001 Nov 2;85(9):1387-95. doi: 10.1054/bjoc.2001.2078.

Abstract

The 67 kD laminin receptor (67LR) binds laminin-1 (LN), major component of the basement membrane, with high affinity. In this study, we demonstrated that human multiple myeloma cell lines (HMCL) and murine 5T2MM cells express 67LR. CD38(bright+) plasma cells in fresh multiple myeloma (MM) bone marrow (BM) samples showed weaker 67LR expression, but expression increased after direct exposure to a BM endothelial cell line (4LHBMEC). LN stimulated the in vitro migration of 3 HMCL (MM5.1, U266 and MMS.1), primary MM cells and the murine 5T2MM cells. 67LR has been shown to mediate the actions of LN through binding to CDPGYIGSR, a 9 amino acid sequence from the B1 chain of LN. MM cell migration was partially blocked by peptide 11, a synthetic nonapeptide derived from this amino sequence and also by a blocking antiserum against 67LR. Co-injection of peptide 11 with 5T2MM cells in a murine in vivo model of MM resulted in a decreased homing of 5T2MM cells to the BM compartment. In conclusion, LN acts as a chemoattractant for MM cells by interaction with 67LR. This interaction might be important during extravasation of circulating MM cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bone Marrow Cells
  • Cell Movement*
  • Chemotactic Factors
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Laminin / pharmacology*
  • Mice
  • Multiple Myeloma / pathology*
  • Neoplastic Cells, Circulating*
  • Receptors, Laminin / physiology*
  • Tumor Cells, Cultured

Substances

  • Chemotactic Factors
  • Laminin
  • Receptors, Laminin
  • laminin 1