Anti-Her-2/neu antibody induces apoptosis in Her-2/neu overexpressing breast cancer cells independently from p53 status

Br J Cancer. 2001 Nov 30;85(11):1764-70. doi: 10.1054/bjoc.2001.2197.

Abstract

Anti-Her-2/neu antibody is known to induce apoptosis in HER-2/neu overexpressing breast cancer cells. However, exact regulatory mechanisms mediating and controlling this phenomenon are still unknown. In the present study, we have investigated the effect of anti-Her-2/neu antibody on apoptosis of HER-2/neu overexpressing human breast cancer cell lines SK-BR-3, HTB-24, HTB-25, HTB-27, HTB-128, HTB-130 and HTB-131 in relation to p53 genotype and bcl-2 status. SK-BR-3, HTB-24, HTB-128 and HTB-130 cells exhibited mutant p53, whereas wild type p53 was found in HTB-25, HTB-27 and HTB-131 cells. All seven cell lines weakly expressed bcl-2 protein (10-20%). Anti-Her-2/neu antibody, irrespective of p53 and bcl-2 status, induced apoptosis in all 7 cell lines dose- and time-dependently and correlated with Her-2/neu overexpression. In addition, incubation of cell lines with anti-Her-2/neu antibody did not alter p53 or bcl-2 expression. Anti-HER-2/neu antibody did not induce apoptosis in HER-2/neu negative HBL-100 and HTB-132 cell lines. Our results indicate that within the panel of tested breast cancer cell lines, anti-Her-2/neu antibody-induced apoptosis was independent from the presence of intact p53.

MeSH terms

  • Antibodies, Monoclonal / pharmacology*
  • Apoptosis / drug effects*
  • Breast Neoplasms / genetics
  • Breast Neoplasms / metabolism
  • Breast Neoplasms / pathology*
  • Cell Cycle / drug effects
  • Cell Division / drug effects
  • DNA Fragmentation / drug effects
  • DNA, Neoplasm / drug effects
  • DNA, Neoplasm / genetics
  • DNA, Neoplasm / metabolism
  • Dose-Response Relationship, Drug
  • Genotype
  • Humans
  • Proto-Oncogene Proteins c-bcl-2 / drug effects
  • Proto-Oncogene Proteins c-bcl-2 / metabolism
  • Receptor, ErbB-2 / immunology
  • Receptor, ErbB-2 / metabolism*
  • Time Factors
  • Tumor Cells, Cultured / drug effects
  • Tumor Suppressor Protein p53 / genetics

Substances

  • Antibodies, Monoclonal
  • DNA, Neoplasm
  • Proto-Oncogene Proteins c-bcl-2
  • Tumor Suppressor Protein p53
  • Receptor, ErbB-2