Deletion of the laminin alpha4 chain leads to impaired microvessel maturation

Mol Cell Biol. 2002 Feb;22(4):1194-202. doi: 10.1128/MCB.22.4.1194-1202.2002.

Abstract

The laminin alpha4 chain, a component of laminin-8 and -9, is expressed in basement membranes, such as those beneath endothelia, the perineurium of peripheral nerves, and around developing muscle fibers. Laminin alpha4-null mice presented with hemorrhages during the embryonic and neonatal period and had extensive bleeding and deterioration of microvessel growth in experimental angiogenesis, as well as mild locomotion defects. Histological examination of newborn mice revealed delayed deposition of type IV collagen and nidogen into capillary basement membranes, and electron microscopy showed discontinuities in the lamina densa. The results demonstrate a central role for the laminin alpha4 chain in microvessel growth and, in the absence of other laminin alpha chains, in the composition of endothelial basement membranes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anemia
  • Animals
  • Animals, Newborn
  • Basement Membrane / chemistry
  • Basement Membrane / ultrastructure
  • Capillaries / embryology
  • Capillaries / growth & development*
  • Capillaries / ultrastructure
  • Collagen / analysis
  • Cornea / blood supply
  • Corneal Neovascularization
  • Fibroblast Growth Factor 2 / pharmacology
  • Hemorrhage
  • Immunohistochemistry
  • Laminin / analysis
  • Laminin / deficiency
  • Laminin / genetics*
  • Laminin / physiology*
  • Membrane Glycoproteins / analysis
  • Mice
  • Mice, Transgenic
  • Muscle, Skeletal / chemistry
  • Protein Isoforms

Substances

  • Lama4 protein, mouse
  • Laminin
  • Membrane Glycoproteins
  • Protein Isoforms
  • nidogen
  • Fibroblast Growth Factor 2
  • Collagen