Liver inflammatory cells, apoptosis, regeneration and stellate cell activation in non-alcoholic steatohepatitis

Dig Liver Dis. 2001 Dec;33(9):768-77. doi: 10.1016/s1590-8658(01)80694-0.

Abstract

Background: The pathogenesis of non-alcoholic steatohepatitis remains unclear from several points of view. Minimal diagnostic criteria are still not defined.

Aim: To gather information useful for diagnosis and to improve the understanding of pathogenic mechanisms.

Patients: A series of 14 patients with non-alcoholic steatohepatitis, identified among liver outpatients, were paired for age, sex and alanine amino transferase values with 14 patients with hepatitis C virus infection without steatosis.

Methods: Clinical, biochemical and immunohistological examination, including characterisation of inflammatory cell population, evaluation of type III collagen and tenascin deposition, activation of stellate cells, hepatocellular apoptosis and proliferation.

Results: Patients with non-alcoholic steatohepatitis were more frequently obese, had higher triglyceride concentrations and lower gamma-globulins. T lymphocytes outnumbered polymorphonuclear cells, both in hepatitis C and in steatohepatitis, with a larger number of CD8 lymphocytes in patients with viral hepatitis but a comparable number of granulocytes. This resulted in a higher granulocytes to T cells ratio in steatohepatitis, possibly making these cells more easily detectable in spite of similar absolute numbers. Portal fibrosis and piecemeal necrosis were prevalent in hepatitis C virus infection, pericentral fibrosis was similar Hepatocellular apoptosis and proliferation as well as stellate cell activation were less relevant in steatohepatitis than in hepatitis C virus infection in spite of similar alanine amino transferase levels.

Conclusions: These data provide a possible explanation for the relatively low tendency to progression of non-alcoholic steatohepatitis in most patients despite increased alanine amino transferase and suggest that non-death-related release of alanine amino transferase might occur in non-alcoholic steatohepatitis. This makes liver biopsy an essential part of the clinical setting supporting diagnosis, evaluation of severity and possibly definition of the evolutionary trend.

MeSH terms

  • Adult
  • Apoptosis
  • Disease Progression
  • Fatty Liver / diagnosis*
  • Fatty Liver / metabolism
  • Fatty Liver / pathology
  • Fatty Liver / physiopathology
  • Female
  • Hepatitis / diagnosis*
  • Hepatitis / metabolism
  • Hepatitis / pathology
  • Hepatitis / physiopathology
  • Humans
  • Immunohistochemistry
  • Lipid Peroxidation
  • Liver / metabolism
  • Liver / pathology
  • Liver Regeneration
  • Male
  • Middle Aged