Ischemic brain tissue salvaged from infarction by the GP IIb/IIIa platelet antagonist tirofiban

Neurology. 2002 Feb 12;58(3):474-6. doi: 10.1212/wnl.58.3.474.

Abstract

In an open pilot study, the authors tested whether the nonpeptide glycoprotein (GP) IIb/IIIa antagonist tirofiban, a highly effective and selective blocker of platelet aggregation, prevents the transition of ischemic brain tissue into the infarct proper as defined by MRI (perfusion-weighted/T2-weighted) in patients with acute ischemic stroke. The infarct volume (T2 lesion after 1 week) was smaller in treated patients (n = 10) compared with matched control subjects (n = 10; p = 0.029) with similar initial perfusion deficit (TTP-maps). The authors conclude that GP IIb/IIIa antagonists have therapeutic potential in acute stroke therapy.

Publication types

  • Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Brain Infarction / drug therapy*
  • Brain Infarction / pathology
  • Brain Infarction / prevention & control
  • Brain Ischemia / drug therapy*
  • Brain Ischemia / pathology
  • Cerebrovascular Circulation / drug effects
  • Female
  • Humans
  • Magnetic Resonance Imaging
  • Male
  • Middle Aged
  • Pilot Projects
  • Platelet Aggregation Inhibitors / administration & dosage*
  • Platelet Glycoprotein GPIIb-IIIa Complex / antagonists & inhibitors*
  • Prospective Studies
  • Tirofiban
  • Tyrosine / administration & dosage*
  • Tyrosine / analogs & derivatives*

Substances

  • Platelet Aggregation Inhibitors
  • Platelet Glycoprotein GPIIb-IIIa Complex
  • Tyrosine
  • Tirofiban