A new C-terminal located mutation (V272ter) in the PIT-1 gene manifesting with severe congenital hypothyroidism. Possible functionality of the PIT-1 C-terminus

Horm Res. 2001;56(3-4):81-6. doi: 10.1159/000048096.

Abstract

Objective: We describe a newborn with clinical signs of severe hypothyroidism and combined pituitary hormone deficiency due to a new mutation in the PIT-1 gene.

Patient and methods: Endocrine stimulation test revealed a deficiency for PRL, TSH and GH, suggesting a defect in the pituitary transcription factor PIT-1. Genetic analysis of the PIT-1 gene was performed by exon-specific PCR, followed by SSCP mutation screening and DNA sequencing of the abnormal migrating fragments.

Results: DNA sequencing revealed a new mutation (V272ter) in direct neighborhood to a known mutational hot spot (R271W) in the C-terminal part of the PIT-1 molecule.

Conclusions: Whereas the R271W mutation has a dominant negative effect on the mutant protein, the newly described mutation is inherited in an autosomal-recessive way. The biological consequences of these two different mutations are discussed.

Publication types

  • Case Reports

MeSH terms

  • Base Sequence / genetics
  • Congenital Hypothyroidism*
  • DNA-Binding Proteins / genetics*
  • Genes, Recessive
  • Humans
  • Hypothyroidism / genetics*
  • Infant
  • Male
  • Mutation / genetics*
  • Polymorphism, Single-Stranded Conformational
  • Transcription Factor Pit-1
  • Transcription Factors / genetics*

Substances

  • DNA-Binding Proteins
  • POU1F1 protein, human
  • Transcription Factor Pit-1
  • Transcription Factors