Defective proliferative responses in B lymphocytes and thymocytes that lack neurofibromin

Mol Immunol. 2002 Feb;38(9):701-8. doi: 10.1016/s0161-5890(01)00101-8.

Abstract

Nf1(-/-) fetal liver cells were used to reconstitute B and T cells in Rag-1(-/-) mice. Lymphocyte development was largely unimpaired in the absence of neurofibromin. However antigen-receptor induced proliferation was defective in neurofibromin deficient peripheral B cells and CD4(+) single positive thymocytes. In contrast to its role as a negative regulator of proliferation in many other cell types, neurofibromin may be a positive regulator of lymphocyte proliferation. Peripheral B cells exhibited circumscribed defects in anti-IgM induced protein tyrosine phosphorylation, which may contribute to the unexpected proliferative defect seen in these cells.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • B-Lymphocytes / pathology
  • B-Lymphocytes / physiology*
  • CD4-Positive T-Lymphocytes / pathology
  • CD4-Positive T-Lymphocytes / physiology*
  • Cell Division / physiology
  • Gene Deletion
  • Lymphocyte Activation / physiology
  • Mice
  • Mice, Knockout
  • Neurofibromin 1 / genetics
  • Neurofibromin 1 / physiology*
  • Signal Transduction / immunology

Substances

  • Neurofibromin 1