Severe CPT-11-induced diarrhea in presence of FK-506 following liver transplantation for hepatocellular carcinoma

Anticancer Res. 2001 Nov-Dec;21(6A):4203-6.

Abstract

Background: The treatment of malignancies in transplanted patients has become an emerging issue. The anticancer agent CPT-11 is hydrolysed to the active metabolite SN-38 and many drugs interact with its metabolism and toxicity.

Patient and methods: We studied the clinical and pharmacological interactions between CPT-11 and FK506 in a liver transplant patient. Serial plasma samples of FK506, CPT-11, SN-38 and SN-38 glucuronide were assayed by high-performance liquid chromatography.

Results: While no CPT-11 toxicity was observed pre-operatively, several post-operative cycles of CPT-11 were complicated with severe diarrhea. No change in FK506 plasma concentrations was noted in the presence of CPT-11 but the pharmacokinetics of CPT-11 was altered in the presence of FK506. SN-38 glucuronidation was reduced for up to 12 hours following CPT-11 infusion. This increase in plasmatic exposure to unbound SN-38 might account for diarrhea.

Conclusion: The starting dose of CPT-11 should be reduced in FK506-treated liver transplant patients.

Publication types

  • Case Reports

MeSH terms

  • Adult
  • Antineoplastic Agents, Phytogenic / adverse effects*
  • Antineoplastic Agents, Phytogenic / blood
  • Antineoplastic Agents, Phytogenic / pharmacokinetics
  • Camptothecin / adverse effects*
  • Camptothecin / analogs & derivatives*
  • Camptothecin / blood
  • Camptothecin / pharmacokinetics
  • Diarrhea / chemically induced*
  • Drug Interactions
  • Glucuronides / blood
  • Glucuronides / pharmacokinetics
  • Humans
  • Irinotecan
  • Liver Transplantation*
  • Tacrolimus / adverse effects*
  • Tacrolimus / blood
  • Tacrolimus / pharmacology

Substances

  • 7-ethyl-10-hydroxycamptothecin glucuronide
  • Antineoplastic Agents, Phytogenic
  • Glucuronides
  • Irinotecan
  • Tacrolimus
  • Camptothecin