Structure of an extended-spectrum class A beta-lactamase from Proteus vulgaris K1

J Mol Biol. 2002 Mar 15;317(1):109-17. doi: 10.1006/jmbi.2002.5420.

Abstract

The structure of a chromosomal extended-spectrum beta-lactamase (ESBL) having the ability to hydrolyze cephalosporins including cefuroxime and ceftazidime has been determined by X-ray crystallography to 1.75 A resolution. The species-specific class A beta-lactamase from Proteus vulgaris K1 was crystallized at pH 6.25 and its structure solved by molecular replacement. Refinement of the model resulted in crystallographic R and R(free) of 16.9 % and 19.3 %, respectively. The folding of the K1 enzyme is broadly similar to that of non-ESBL TEM-type beta-lactamases (2 A rmsd for C(alpha)) and differs by only 0.35 A for all atoms of six conserved residues in the catalytic site. Other residues promoting extended-spectrum activity in K1 include the side-chains of atypical residues Ser237 and Lys276. These side-chains are linked by two water molecules, one of which lies in the position normally filled by the guanidinium group of Arg244, present in most non-ESBL enzymes but absent from K1. The ammonium group of Lys276, ca 3.5 A from the virtual Arg244 guanidinium position, may interact with polar R2 substitutents on the dihydrothiazene ring of cephalosporins.

MeSH terms

  • Binding Sites
  • Crystallography, X-Ray
  • Drug Resistance, Bacterial
  • Hydrogen Bonding
  • Models, Molecular
  • Protein Conformation
  • Proteus vulgaris / classification*
  • Proteus vulgaris / enzymology*
  • beta-Lactamases / chemistry*
  • beta-Lactamases / classification*
  • beta-Lactamases / metabolism

Substances

  • beta-Lactamases

Associated data

  • PDB/1HZO