Macrolide and tetracycline resistance and molecular relationships of clinical strains of Streptococcus agalactiae

Antimicrob Agents Chemother. 2002 May;46(5):1574-6. doi: 10.1128/AAC.46.5.1574-1576.2002.

Abstract

Mechanisms for tetracycline and macrolide resistance in 54 isolates of erythromycin-resistant Streptococcus agalactiae were analyzed by PCR. The erm(B), erm(A), and mef(A) genes, either alone or in combination, were detected in all the erythromycin-resistant isolates. The tet(M) and tet(O) genes were responsible for tetracycline resistance. Random amplification of polymorphic DNA indicated different clonal origins of the isolates.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anti-Bacterial Agents / pharmacology*
  • Bacterial Proteins / genetics
  • Drug Resistance, Bacterial / genetics*
  • Erythromycin / pharmacology*
  • Humans
  • Microbial Sensitivity Tests
  • Polymerase Chain Reaction
  • Random Amplified Polymorphic DNA Technique
  • Streptococcal Infections / microbiology
  • Streptococcus agalactiae / drug effects*
  • Streptococcus agalactiae / genetics
  • Tetracycline / pharmacology*
  • Tetracycline Resistance / genetics

Substances

  • Anti-Bacterial Agents
  • Bacterial Proteins
  • Erythromycin
  • Tetracycline