Nitric oxide modulation of renal and cardiac hemodynamics in type 2 diabetes

Eur J Endocrinol. 2002 May;146(5):687-94. doi: 10.1530/eje.0.1460687.

Abstract

Objective: To evaluate endothelial function in type 2 diabetic patients with and without diabetic nephropathy.

Methods: We studied the effects of systemic infusion of the nitric oxide (NO) synthase inhibitor NG-monomethyl-l-arginine (L-NMMA) on cardiovascular and renal hemodynamics in six type 2 diabetic patients with microalbuminuria (D2-MA), six type 2 diabetic patients with normoalbuminuria (D2-NA) and five control subjects. Both type 2 diabetic patients and control subjects had mild arterial hypertension.

Results: L-NMMA infusion decreased the cardiac index in all groups. A reduction in glomerular filtration rate (GFR) and an increase in filtration fraction were observed only in controls. Renal plasma flow decreased in controls and D2-NA patients and renal vascular resistance increased in all groups.

Conclusions: The effect of L-NMMA on cardiac output was similar in controls and type 2 diabetic patients with and without diabetic nephropathy. In contrast, the effect on GFR was impaired in both diabetic groups, suggesting that glomerular NO homeostasis is altered in type 2 diabetes. Moreover the discrepancy, in diabetic patients, between cardiac and renal effects during L-NMMA infusion suggests that the modulation of glomerular hemodynamics is independent from NO-regulated cardiac output.

Publication types

  • Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Albuminuria / etiology
  • Cardiac Output / drug effects
  • Coronary Circulation / physiology*
  • Diabetes Mellitus, Type 2 / physiopathology*
  • Diabetes Mellitus, Type 2 / urine
  • Diabetic Nephropathies / physiopathology
  • Enzyme Inhibitors / pharmacology
  • Glomerular Filtration Rate / drug effects
  • Hemodynamics
  • Humans
  • Hypertension / physiopathology
  • Male
  • Middle Aged
  • Nitric Oxide / physiology*
  • Reference Values
  • Renal Circulation / physiology*
  • omega-N-Methylarginine / pharmacology

Substances

  • Enzyme Inhibitors
  • omega-N-Methylarginine
  • Nitric Oxide