Encapsulated plasmid DNA treatment for human papillomavirus 16-associated anal dysplasia: a Phase I study of ZYC101

Clin Cancer Res. 2002 May;8(5):1028-37.

Abstract

High-grade dysplasia induced by high-risk types of human papillomavirus (HPV) precedes invasive cancer in anal squamous epithelium just as it does in the cervix. A therapeutic HPV vaccine strategy as a potential treatment for anal dysplasia was tested in a standard Phase I dose escalation trial. The primary objective was to evaluate the safety of the agent; additional study aims were to evaluate the histological response, immune response, and effect on anal HPV-16 infection. Each subject was treated with four i.m. injections of 50-400 microg of ZYC101 at 3-week intervals. ZYC101 is composed of plasmid DNA encapsulated in biodegradable polymer microparticles. The plasmid DNA encodes for multiple HLA-A2-restricted epitopes derived from the HPV-16 E7 protein, one of two HPV oncoproteins consistently expressed in neoplastic cells. Fifty-six potential anal dysplasia subjects were screened to identify 12 eligible subjects with HPV-16 anal infection and a HLA-A2 haplotype. The investigational agent was well tolerated in all subjects at all dose levels tested. Three subjects experienced partial histological responses, including one of three subjects receiving the 200-microg dose and two subjects at the 400-microg dose level. Using a direct Elispot, 10 of 12 subjects demonstrated increased immune response to the peptide epitopes encoded within ZYC101; each continued to show elevated immune responses 6 months after the initiation of therapy. These results support the continued investigation of a therapeutic vaccination strategy for anal dysplasia.

Publication types

  • Clinical Trial
  • Clinical Trial, Phase I
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • Aged
  • Anus Neoplasms / drug therapy
  • Anus Neoplasms / immunology*
  • Anus Neoplasms / virology
  • DNA, Viral / drug effects
  • DNA, Viral / genetics
  • DNA, Viral / metabolism
  • Dose-Response Relationship, Drug
  • Erythema / chemically induced
  • Fatigue / chemically induced
  • Female
  • Fever / chemically induced
  • HLA-A2 Antigen / immunology
  • Headache / chemically induced
  • Humans
  • Male
  • Microspheres
  • Middle Aged
  • Oncogene Proteins, Viral / genetics
  • Oncogene Proteins, Viral / immunology*
  • Oncogene Proteins, Viral / therapeutic use
  • Pain / chemically induced
  • Papillomaviridae / genetics
  • Papillomaviridae / growth & development
  • Papillomaviridae / immunology
  • Papillomavirus E7 Proteins
  • Papillomavirus Infections / drug therapy
  • Papillomavirus Infections / immunology*
  • Papillomavirus Infections / virology
  • Peptide Fragments / genetics
  • Peptide Fragments / immunology
  • Peptide Fragments / therapeutic use
  • Plasmids / administration & dosage
  • Plasmids / genetics
  • Time Factors
  • Treatment Outcome
  • Tumor Virus Infections / diagnosis
  • Tumor Virus Infections / drug therapy
  • Tumor Virus Infections / immunology*
  • Vaccines, DNA / adverse effects
  • Vaccines, DNA / immunology
  • Vaccines, DNA / therapeutic use

Substances

  • DNA, Viral
  • HLA-A2 Antigen
  • Oncogene Proteins, Viral
  • Papillomavirus E7 Proteins
  • Peptide Fragments
  • Vaccines, DNA
  • oncogene protein E7, Human papillomavirus type 16